Monitor ABG (less than 12% is still considered normal).Assess for signs of acute respiratory distress syndrome (rales and/or wheezes).Assess for other neurologic and other systemic signs like:.Monitor signs of cerebral hypoxia (confusion), for it has the possibility of rapid progression to coma. ![]() Recheck for the level of consciousness.Be alert for altered breathing patterns and episodes of apnea.Expect for elevated respiratory and pulse rates.Determine the client’s underlying health status that would cause higher risk, especially for the presence of anemia, pulmonary disease, and/or cardiac disease.Gather incident history from the patient or any person, particularly the type and length of exposure.Monitor for signs on the necessity for intubation.Make sure the mask fits the client’s face to deliver the desired amount. Position to semi-Fowler’s if not contraindicated.The client may manifest respiratory depression (5-10 per minute). Muscles around air passages may relax if the client turned unconscious due to prolonged exposure or massive poisoning. Check for airway obstruction if the client is unconscious.This is due to the formation of laryngeal edema from thermal injury. If it is due to carbon monoxide smoke inhalation, stridor may be assessed. It primarily causes tissue anoxia, which later leads to more severe health problems, and worst, death. It may be in the form of accidental inhalation or intentionally inflicted like that of suicide. ![]() It may occur at home or in industrial places. Its affinity to hemoglobin is 200-300 times than that of the oxygen-hemoglobin.Ĭarbon Monoxide poisoning is a type of inhalation poisoning through overexposure to carbon monoxide. ![]() It has the capacity to bind with the circulating blood hemoglobin, producing carboxyhemoglobin which reduces the oxygen-carrying capacity of the blood. Carbon Monoxide is a gas product of incomplete hydrocarbon combustion.
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